Publications

Descriptives per condition. We implemented experimental conditions in eight time slots. SDSs were only handed out during these times. Six experimental conditions lasted two hours, while two lasted one hour. Except in the buzzer conditions, the SDS was covered in a black bag so the flashing light was invisible.

This is the adjacency matrix containing the empirical weight parameters that were used as a starting point for the simulation study. These parameters were retrieved from a network analysis with IsingFit in R, the data come from the VATSPUD study (courtesy of Ken Kendler, see the paper for a brief description of the data) and consist of the 14 disaggregated symptoms of major depression according to DSM-III-R.

A fundamental question in psychotherapy is whether interventions should target client problems (i.e., problem-focused approaches) or client strengths (i.e., strength-focused approaches). In this study, we first propose to address this question from a network perspective on schema modes (i.e., healthy or dysfunctional patterns of co-occurring emotions, cognitions, and behaviours). From this perspective, schema modes mutually influence each other (e.g., healthy modes reduce dysfunctional modes). Recent evidence suggests that changes in modes that are strongly associated to other modes (i.e., central modes) could be associated with greater treatment effects. We therefore suggest research should investigate the relative centrality of healthy and dysfunctional modes. To make an exploratory start, we investigated the cross-sectional network structure of schema modes in a clinical (comprising individuals diagnosed with paranoid, narcissistic, histrionic, and Cluster C personality disorders) and non-clinical sample. Results showed that, in both samples, the Healthy Adult was significantly less central than several dysfunctional modes (e.g., Undisciplined Child and Abandoned and Abused Child). Although our study cannot draw causal conclusions, this finding could suggest that weakening dysfunctional modes (compared to strengthening the Healthy Adult) might be more effective in decreasing other dysfunctional modes. Our study further indicates that several schema modes are negatively associated, which could suggest that decreasing one might increase another. Finally, the Healthy Adult was among the modes that most strongly discriminated between clinical and non-clinical individuals. Longitudinal and experimental research into the network structure of schema modes is required to further clarify the relative influence of schema modes.

In this paper, we characterize major depression (MD) as a complex dynamic system in which symptoms (e.g., insomnia and fatigue) are directly connected to one another in a network structure. We hypothesize that individuals can be characterized by their own network with unique architecture and resulting dynamics. With respect to architecture, we show that individuals vulnerable to developing MD are those with strong connections between symptoms: e.g., only one night of poor sleep suffices to make a particular person feel tired. Such vulnerable networks, when pushed by forces external to the system such as stress, are more likely to end up in a depressed state; whereas networks with weaker connections tend to remain in or return to a non-depressed state. We show this with a simulation in which we model the probability of a symptom becoming 'active' as a logistic function of the activity of its neighboring symptoms. Additionally, we show that this model potentially explains some well-known empirical phenomena such as spontaneous recovery as well as accommodates existing theories about the various subtypes of MD. To our knowledge, we offer the first intra-individual, symptom-based, process model with the potential to explain the pathogenesis and maintenance of major depression.

In psychological measurement, two interpretations of measurement systems have been developed: the reflective interpretation, in which the measured attribute is conceptualized as the common cause of the observables, and the formative interpretation, in which the measured attribute is seen as the common effect of the observables. We advocate a third interpretation, in which attributes are conceptualized as systems of causally coupled (observable) variables. In such a view, a construct like ’depression’ is not seen as a latent variable that underlies symptoms like ’lack of sleep’ or ’fatigue’, and neither as a composite constructed out of these symptoms, but as a system of causal relations between the symptoms themselves (e.g., lack of sleep → fatigue, etc.). We discuss methodological strategies to investigate such systems as well as theoretical consequences that bear on the question in which sense such a construct could be interpreted as real.

We outline an array of journal policies that JPSP:ASC could adopt to further promote transparent and responsible research practices; in turn, these practices will increase the reliability of research findings published in JPSP:ASC.

The network perspective on psychopathology understands mental disorders as complex networks of interacting symptoms. Despite its recent debut, with conceptual foundations in 2008 and empirical foundations in 2010, the framework has received considerable attention and recognition in the last years.