THE EFFECTS OF BLOCKING CALCITONIN GENE RELATED PEPTIDE, and NITRIC OXIDE SYNTHASE IN A RAT MODEL OF ENDOTOXIC SHOCK

SHOCK®: Injury, Inflammation, and Sepsis: Laboratory and Clinical Approaches includes studies of novel therapeutic approaches, such as immunomodulation, gene therapy, nutrition, and others. The mission of the Journal is to foster and promote multidisciplinary studies, both experimental and clinical in nature, that critically examine the etiology, mechanisms and novel therapeutics of shock-related pathophysiological conditions. Its purpose is to excel as a vehicle for timely publication in the areas of basic and clinical studies of shock, trauma, sepsis, inflammation, ischemia, and related pathobiological states, with particular emphasis on the biologic mechanisms that determine the response to such injury. Making such information available will ultimately facilitate improved care of the traumatized or septic individual. SHOCK® is also privileged to have fourteen distinguished Associate Editors, as well as a diverse group of editorial board members from various parts of the world, who enjoy the respect and admiration of their colleagues in their areas of investigation. The Official Journal of the Shock Society, the European Shock Society, the Indonesian Shock Society, the International Federation of Shock Societies, and the Official and International Journal of the Japan Shock Society