Retinal And Choroidal Neovascularization, Blood-retinal Barrier Breakdown, And Apoptosis Are Inhibited In The Absence Of Prolyl Hydroxylase-1

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Ischemia Induces Blood-retinal Barrier (BRB) Breakdown through HIF-1 Dependent VEGF Upregulation and VEGF Mediates BRB Breakdown through ICAM-1 and Adenosine

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Prolyl hydroxylase-1 regulates hepatocyte apoptosis in an NF-κB-dependent manner

Susan F. Fitzpatrick, Zsolt Fábián, Bettina Schaible, Colin R. Lenihan, Thomas Schwarzl, Javier Rodríguez, Xingnan Zheng, Zongwei Li, Murtaza M. Tambuwala, Desmond G. Higgins, Yvonne O’Meara, Craig Slattery, Mario C. Manresa, Peter Fraisl, Ulrike Brüning, Myriam Baes, Peter Carmeliet, Glen Doherty, Alex von Kriegsheim, Eoin P. Cummins, Cormac T. Taylor

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Prolyl hydroxylase-1 regulates hepatocyte apoptosis in an NF-κB-dependent manner

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Implication of the hypoxia response element of the vegf promoter in mouse models of retinal and choroidal neovascularization, but not retinal vascular development

Stanley A. Vinores, Wei‐Hong Xiao, Sadia Aslam, Jikui Shen, Yuji Oshima, Hiroyuki Nambu, Hansheng Liu, Peter Carmeliet, Peter A. Campochiaro

Retinal And Choroidal Neovascularization, Blood-retinal Barrier Breakdown, And Apoptosis Are Inhibited In The Absence Of Prolyl Hydroxylase-1

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Loss of Prolyl Hydroxylase-1 Protects Against Colitis Through Reduced Epithelial Cell Apoptosis and Increased Barrier Function

Ischemia Induces Blood-retinal Barrier (BRB) Breakdown through HIF-1 Dependent VEGF Upregulation and VEGF Mediates BRB Breakdown through ICAM-1 and Adenosine

Prolyl hydroxylase-1 regulates hepatocyte apoptosis in an NF-κB-dependent manner

Prolyl hydroxylase-1 regulates hepatocyte apoptosis in an NF-κB-dependent manner

Implication of the hypoxia response element of the vegf promoter in mouse models of retinal and choroidal neovascularization, but not retinal vascular development