Podocyte Repopulation Contributes to Regression of Glomerular Injury Induced by Ace Inhibition

Daniela Macconi; Fabio Sangalli; Maria Bonomelli; Sara Conti; Lucia Condorelli; Elena Gagliardini; Giuseppe Remuzzi; Andrea Remuzzi

doi:10.2353/ajpath.2009.080227

Discussion(0)

No comments yet. Be the first to comment.

Related publications

Article2017

BRAF Signaling Pathway Inhibition, Podocyte Injury, and Nephrotic Syndrome

Luca Perico, Mario Mandalà, Arrigo Schieppati, Camillo Carrara, Paola Rizzo, Sara Conti, Lorena Longaretti, Ariela Benigni, Giuseppe Remuzzi

Article2006

Pathophysiologic Implications of Reduced Podocyte Number in a Rat Model of Progressive Glomerular Injury

Daniela Macconi, Maria Bonomelli, Ariela Benigni, Tiziana Plati, Fabio Sangalli, Lorena Longaretti, Sara Conti, Hiroshi Kawachi, Prue Hill, Giuseppe Remuzzi, Andrea Remuzzi

Article1999

Glomerular size-selective dysfunction in NIDDM is not ameliorated by ACE inhibition or by calcium channel blockade

Piero Ruggenenti, Lidia Mosconi, Fabio Sangalli, Federica Casiraghi, Vincenzo Gambara, Giuseppe Remuzzi, Andrea Remuzzi

Article2004

Changes in glomerular perm-selectivity induced by angiotensin II imply podocyte dysfunction and slit diaphragm protein rearrangement

Ariela Benigni, Elena Gagliardini, Giuseppe Remuzzi

Article1991

Nature and extent of glomerular injury induced by cyclosporine in heart transplant patients

Tullio Bertani, Paolo Ferrazzi, Arrigo Schieppati, Piero Ruggenenti, Amando Gamba, L Parenzan, G Mecca, Norberto Perico, O Imberti, Andrea Remuzzi, Giuseppe Remuzzi