Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: Implications for the pathways to chronic depression and neuroprogression

Michael Maes; Marta Kubera; Ivana Mihaylova; M. Geffard; Piotr Gałecki; Jean-Clude Leunis; Michael Berk

doi:10.1016/j.jad.2012.06.039

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Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: Implications for the pathways to chronic depression and neuroprogression — Michael Maes (2012) | RDL Network

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Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: Implications for the pathways to chronic depression and neuroprogression

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Michael Maes

University of Electronic Science and Technology of China

Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: Implications for the pathways to chronic depression and neuroprogression

Article 2012 en

Authors

Michael Maes
University Of Electronic Science & Technology Of China
MK
Marta Kubera
IM
Ivana Mihaylova

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Publication Info

DOI: 10.1016/j.jad.2012.06.039
Year: 2012
Published: —
Language: en

Article Details

Volume: 149
Issue: 1-3
Link Of The Paper: https://doi.org/10.1016/j.jad.2012.06.039

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Created:

June 19, 2026

Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: Implications for the pathways to chronic depression and neuroprogression

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In depression, bacterial translocation may drive inflammatory responses, oxidative and nitrosative stress (O&amp;NS), and autoimmune responses directed against O&amp;NS‐damaged neoepitopes

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