Phosphorus is a mineral element essential to cellular function. The prevalence of hypophosphatemia is particularly high in critically ill patients. Hypophosphatemia may be the result of increased renal excretion, reduced digestive absorption, intracellular shift accompanied or not by depletion of the total phosphorus pool, and extreme catabolic states. Clinical manifestations are especially apparent in severe hypophosphatemia and include impairments of the respiratory, musculo-skeletal, pulmonary, and neurological systems. Hypophosphatemia must be corrected, especially if it is severe and symptomatic.
Marlene Panzer, Eva Meindl, Benedikt Schaefer, Sonja Wagner, Bernhard Glodny, Gert Mayer, Andreas Pircher, Christoph Schwarz, Felix Beckmann, Clivia Hejny, Bastian Joachim, Juergen Konzett, Herbert Tilg, Isabel Heidegger, Myles Wolf, Ralf Weiskirchen, Heinz Zoller
Berkan Kurt, Lukas Buendgens, Theresa H. Wirtz, Sven H. Loosen, Maximilian Schulze‐Hagen, Daniel Truhn, Jonathan F. Brozat, Samira Abu Jhaisha, Philipp Hohlstein, Ger H. Koek, Ralf Weiskirchen, Christian Trautwein, Frank Tacke, Karim Hamesch, Alexander Koch
Alexander Koch, Ralf Weiskirchen, Alexander Krusch, Jan Bruensing, Lukas Buendgens, Ulf Herbers, Eray Yagmur, Ger H. Koek, Christian Trautwein, Frank Tacke
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