Familial clustering of MR exists in the community, supporting a genetic susceptibility common to primary and nonprimary MR. Further studies are needed to elucidate the common regulatory pathways that may lead to MR irrespective of its cause.
Nicolas M. Van Mieghem, Nicolò Piazza, Robert H. Anderson, Apostolos Tzikas, Koen Nieman, Lotte E. de Groot–de Laat, Jackie S. McGhie, Marcel L. Geleijnse, Ted Feldman, Patrick W. Serruys, Peter P. de Jaegere
Lu‐Chen Weng, Seung Hoan Choi, Derek Klarin, Po−Ru Loh, Mark Chaffin, Carolina Roselli, Olivia Hulme, J. G. Smith, Sekar Kathiresan, Kathryn L. Lunetta, Josée Dupuis, Chris Newton-Cheh, Emelia Benjamin, Patrick T. Ellinor, Steven A. Lubitz
Eskil Kreiner, Johannes Waage, Marie Standl, Susanne Brix, Tune H. Pers, Alexessander Couto Alves, Nicole M. Warrington, Carla M. T. Tiesler, Elaine Fuertes, Lude Franke, Joel N. Hirschhorn, Anthony James, Angela Simpson, Joyce Y. Tung, Gerard H. Koppelman, Dirkje S. Postma, Craig E. Pennell, Paul M Ridker, Adnan Ćustović, Nicholas J. Timpson, Manuel A. R. Ferreira, David P. Strachan, John Henderson, David A. Hinds,
Eskil Kreiner, Johannes Waage, Marie Standl, Susanne Brix, Tune H. Pers, Alexessander Couto Alves, Nicole M. Warrington, Carla M. T. Tiesler, Elaine Fuertes, Lude Franke, Joel N. Hirschhorn, Anthony James, Angela Simpson, Joyce Y. Tung, Gerard H. Koppelman, Dirkje S. Postma, Craig E. Pennell, Paul M Ridker, Adnan Ćustović, Nicholas J. Timpson, Manuel A. R. Ferreira, David P. Strachan, John Henderson, David A. Hinds,
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