Brachial Artery Vasodilator Function and Systemic Inflammation in the Framingham Offspring Study
Article 2004 en
Authors
JV
Joseph A. Vita
JK
John F. Keaney
ML
Martin G. Larson
Abstract
1 min read
Our observations are consistent with the hypothesis that risk factors induce a state of inflammation that impairs vascular function. For flow-mediated dilation, we found no evidence that inflammation has additional effects beyond those attributable to traditional risk factors. The incremental contribution of CRP, IL-6, and sICAM-1 to reactive hyperemia above and beyond known risk factors suggests that systemic inflammation may contribute to impaired vasomotor function in forearm microvessels.
Gary F. Mitchell, Helen Parise, Joseph A. Vita, Martin G. Larson, Elaine Warner, John F. Keaney, Michelle J. Keyes, Daniel Levy, Ramachandran S. Vasan, Emelia Benjamin
Leroy L. Cooper, Solomon K. Musani, Floyd Washington, Jonathan P. Moore, Avnish Tripathi, Connie W. Tsao, Naomi M. Hamburg, Emelia Benjamin, Ramachandran S. Vasan, Gary F. Mitchell, Ervin R. Fox
Nisha I. Parikh, Michelle J. Keyes, Martin G. Larson, Karla M. Pou, Naomi M. Hamburg, Joseph A. Vita, Christopher J. O’Donnell, Ramachandran S. Vasan, Gary F. Mitchell, Udo Hoffmann, Caroline S. Fox, Emelia Benjamin
Gary F. Mitchell, Joseph A. Vita, Martin G. Larson, Helen Parise, Michelle J. Keyes, Elaine Warner, Ramachandran S. Vasan, Daniel Levy, Emelia Benjamin
Discussion(0)
No comments yet. Be the first to comment.