β-catenin and its alteration in an experimental model of diabetic nephropathy.
Article 2014 en
Authors
SP
Se Jin Park
EA
Eun Mi Ahn
TH
Tae Sun Ha
Abstract
1 min read
Our results suggest that AGE and hyperglycemia may induce the cytoplasmic redistribution of beta-catenin and inhibit the production of beta-catenin at the transcriptional and posttranslational levels, which may result in the development of kidney dysfunction in diabetic conditions.
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